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A 3-year-old boy developed avascular bone necrosis of the femoral head (AVNF) at the affected site after exposure to steroids for treatment of upper airway obstruction. GUCY2C polymorphism was noted in the patient, but not in his mother, suggesting a possible genetic role in the pathogenesis of the disease. Exome and genome sequencing were performed using DNA extracted from the patient’s and mother’s blood samples. Homozygous variants, c.111G>C (p.Asp37His) in GUCY2C and c.811C>T (p.Pro271Leu) in H3F3A were identified in the patient; neither had been previously reported. The patient was the only one of 14 family members to be affected by AVNF, and a de novo mutation in GUCY2C seemed to be associated with the disease. The patient was homozygous for c.811C>T (p.Pro271Leu) in H3F3A, and this variant was suggested to be a potential risk factor for AVNF. Our findings suggest that the bone metabolism of children with GUCY2C variants may be altered, and they may therefore be more susceptible to avascular bone necrosis, although the precise mechanism is unknown.Welcome to the home of Disneyland!
First, let’s set some context.
Disneyland is a resort owned by The Walt Disney Company and is located in Anaheim, California.
The theme park was formerly “Disneyland Park” and opened on July 17, 1955. The park has grown to include five attractions, two hotels, and over 40 restaurants.
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